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dc.contributor.authorCendán, Cruz Miguel-
dc.contributor.authorPujalte, José Miguel-
dc.contributor.authorPortillo-Salido, Enrique-
dc.contributor.authorMontoliu, Lluís-
dc.contributor.authorBaeyens, José M.-
dc.date.accessioned2008-12-03T11:57:48Z-
dc.date.available2008-12-03T11:57:48Z-
dc.date.issued2005-03-09-
dc.identifier.citationEuropean Journal of Pharmacology 511(1): 73-74 (2005)en_US
dc.identifier.issn0014-2999-
dc.identifier.urihttp://hdl.handle.net/10261/8965-
dc.description2 pages, 1 figure.-- PMID: 15777781 [PubMed]en_US
dc.description.abstractThe role of σ1 receptors in non-acute pain has not been explored. In this study we show that both phases of formalin-induced pain were reduced by approximately 55% in σ1 receptor knockout mice in comparison to wild-type animals. These results suggest that the tonic pain induced by formalin is altered in mice lacking σ1 receptors, and highlight the potential usefulness of further studies of the role of σ1 receptors in models of non-acute pain.en_US
dc.description.sponsorshipThe experiments performed in the laboratory headed by José M. Baeyens are supported in part by a grant from Junta de Andalucía (CTS 109).en_US
dc.format.extent86993 bytes-
dc.format.mimetypeapplication/pdf-
dc.language.isoengen_US
dc.publisherElsevieren_US
dc.rightsclosedAccessen_US
dc.subjectσ1 Receptoren_US
dc.subjectFormalinen_US
dc.subjectNon-acute painen_US
dc.titleFormalin-induced pain is reduced in σ1 receptor knockout miceen_US
dc.typeartículoen_US
dc.identifier.doihttp://dx.doi.org/10.1016/j.ejphar.2005.01.036-
dc.description.peerreviewedPeer revieweden_US
dc.relation.publisherversionhttp://dx.doi.org/10.1016/j.ejphar.2005.01.036en_US
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