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Campo DC | Valor | Lengua/Idioma |
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dc.contributor.author | Kim, Woe-Yeon | - |
dc.contributor.author | Yun, Dae-Jin | - |
dc.contributor.author | Pérez Hormaeche, J. | - |
dc.contributor.author | Quintero, Francisco J. | - |
dc.contributor.author | Pardo, José M. | - |
dc.date.accessioned | 2013-01-22T12:00:29Z | - |
dc.date.available | 2013-01-22T12:00:29Z | - |
dc.date.issued | 2013-01-15 | - |
dc.identifier.citation | Nature Communications 4 Article number 1352: (2013) | es_ES |
dc.identifier.uri | http://hdl.handle.net/10261/64691 | - |
dc.description | Kim, Woe-Yeon et al.-- | es_ES |
dc.description.abstract | Environmental challenges to plants typically entail retardation of vegetative growth and delay or cessation of flowering. Here we report a link between the flowering time regulator, GIGANTEA (GI), and adaptation to salt stress that is mechanistically based on GI degradation under saline conditions, thus retarding flowering. GI, a switch in photoperiodicity and circadian clock control, and the SNF1-related protein kinase SOS2 functionally interact. In the absence of stress, the GI:SOS2 complex prevents SOS2- based activation of SOS1, the major plant Na+/H+-antiporter mediating adaptation to salinity. GI over-expressing, rapidly flowering, plants show enhanced salt sensitivity, whereas gi mutants exhibit enhanced salt tolerance and delayed flowering. Salt-induced degradation of GI confers salt tolerance by the release of the SOS2 kinase. The GISOS2 interaction introduces a higher order regulatory circuit that can explain in molecular terms, the long observed connection between floral transition and adaptive environmental stress tolerance in Arabidopsis. | es_ES |
dc.description.sponsorship | This research was supported by the Next-Generation BioGreen 21 Program (Systems and Synthetic Agrobiotech Center, no. PJ008025), a Cooperative Research Program for Agriculture Science & Technology Development (Project No. PJ007850), and the Ministry of Education, Science and Technology for the World Class University (WCU) program (R32-10148) from the Rural Development Administration, Republic of Korea, and by grant BIO2009-08641 financed by the Spanish Ministry of Science and Innovation and the FEDER program. | es_ES |
dc.language.iso | eng | es_ES |
dc.publisher | Nature Publishing Group | es_ES |
dc.rights | openAccess | es_ES |
dc.title | Release of SOS2 kinase from sequestration with GIGANTEA determines salt tolerance in Arabidopsis | es_ES |
dc.type | artículo | es_ES |
dc.identifier.doi | 10.1038/ncomms2357 | - |
dc.description.peerreviewed | Peer reviewed | es_ES |
dc.relation.publisherversion | http://dx.doi.org/10.1038/ncomms2357 | es_ES |
dc.identifier.e-issn | 2041-1723 | - |
dc.type.coar | http://purl.org/coar/resource_type/c_6501 | es_ES |
item.openairetype | artículo | - |
item.grantfulltext | open | - |
item.cerifentitytype | Publications | - |
item.openairecristype | http://purl.org/coar/resource_type/c_18cf | - |
item.fulltext | With Fulltext | - |
item.languageiso639-1 | en | - |
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Release of SOS2 kinase from sequestration with GIGANTEA determines salt tolerance in Arabidopsis.pdf | 1,63 MB | Adobe PDF | Visualizar/Abrir |
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