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dc.contributor.authorHernández-Allés, Santiago-
dc.contributor.authorBenedí Benito, Vicente Javier-
dc.contributor.authorMartínez-Martínez, Luis-
dc.contributor.authorPascual, Álvaro-
dc.contributor.authorAguilar, Alicia-
dc.contributor.authorTomás, Juan M.-
dc.contributor.authorAlbertí, Sebastián-
dc.date.accessioned2012-07-24T10:44:38Z-
dc.date.available2012-07-24T10:44:38Z-
dc.date.issued1999-
dc.identifierissn: 0066-4804-
dc.identifier.citationAntimicrobial Agents and Chemotherapy 43: 937-939 (1999)-
dc.identifier.urihttp://hdl.handle.net/10261/53833-
dc.description.abstractWe have demonstrated by using an in vitro approach that interruption of the OmpK36 porin gene by insertion sequences (ISs) is a common type of mutation that causes loss of porin expression and increased resistance to cefoxitin in Klebsiella pneumoniae. This mechanism also operates in vivo: of 13 porin-deficient cefoxitin-resistant clinical isolates of K. pneumoniae, 4 presented ISs in their ompK36 gene.-
dc.language.isoeng-
dc.publisherAmerican Society for Microbiology-
dc.rightsclosedAccess-
dc.titleDevelopment of resistance during antimicrobial therapy caused by insertion sequence interruption of porin genes-
dc.typeartículo-
dc.date.updated2012-07-24T10:44:38Z-
dc.description.versionPeer Reviewed-
Appears in Collections:(IMEDEA) Artículos
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