English   español  
Please use this identifier to cite or link to this item: http://hdl.handle.net/10261/52958
Share/Impact:
Statistics
logo share SHARE logo core CORE   Add this article to your Mendeley library MendeleyBASE

Visualizar otros formatos: MARC | Dublin Core | RDF | ORE | MODS | METS | DIDL
Exportar a otros formatos:
DC FieldValueLanguage
dc.contributor.authorHeredia, Fabiana-
dc.contributor.authorNieto, M. Ángela-
dc.date.accessioned2012-07-09T07:58:19Z-
dc.date.available2012-07-09T07:58:19Z-
dc.date.issued2011-
dc.identifierdoi: 10.1016/j.stem.2011.04.011-
dc.identifierissn: 1934-5909-
dc.identifiere-issn: 1875-9777-
dc.identifier.citationCell Stem Cell 8(5): 462-463 (2011)-
dc.identifier.urihttp://hdl.handle.net/10261/52958-
dc.description.abstractEpithelial plasticity is crucial during embryonic development and progression of carcinomas. In this issue, Abell et al. (2011) show that acetylation on histones H2A/H2B prevents epithelial cells from undergoing epithelial-to-mesenchymal transition (EMT). Their findings also suggest that under conditions where EMT and stemness coexist, they can be independently regulated. © 2011 Elsevier Inc.-
dc.language.isoeng-
dc.publisherCell Press-
dc.rightsclosedAccess-
dc.titleAn epigenetic mark that protects the epithelial phenotype in health and disease-
dc.typeartículo-
dc.identifier.doi10.1016/j.stem.2011.04.011-
dc.date.updated2012-07-09T07:58:19Z-
dc.description.versionPeer Reviewed-
Appears in Collections:(IN) Artículos
Files in This Item:
File Description SizeFormat 
accesoRestringido.pdf15,38 kBAdobe PDFThumbnail
View/Open
Show simple item record
 

Related articles:


WARNING: Items in Digital.CSIC are protected by copyright, with all rights reserved, unless otherwise indicated.