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dc.contributor.authorBadimón, Lina-
dc.contributor.authorVilahur, Gemma-
dc.date.accessioned2008-05-09T14:31:17Z-
dc.date.available2008-05-09T14:31:17Z-
dc.date.issued2007-11-01-
dc.identifier.citationCerebrovasc Dis. 24 Suppl 1: 30-39 (2007).en_US
dc.identifier.issn1015-9770-
dc.identifier.urihttp://hdl.handle.net/10261/4130-
dc.descriptionPMID: 17971637.-- Final full-text version available at: http://dx.doi.org/10.1159/000107377en_US
dc.description.abstractAtherosclerosis is a systemic disease that starts early in life, asymptomatically progressing though adulthood, until clinically manifested. In the last few years, experimental, clinical and pathological investigation has led us to a better knowledge of the pathophysiology of atherothrombotic disease. Atherothrombosis is the result of atherosclerosis progression, and its potentially life-threatening clinical consequences include coronary artery disease, cerebrovascular disease and peripheral artery disease. These events are mostly secondary to atherosclerotic plaque disruption and subsequent in situ thrombus formation which may be embolized and dragged by the arterial flow until occluding distal vessels. The demonstrated beneficial role of antiplatelet drugs in reducing the incidence of nonfatal events in many large clinical trials has demonstrated the major role of platelets in the atherothrombotic process in the arterial tree.en_US
dc.format.extent22195 bytes-
dc.format.mimetypeapplication/pdf-
dc.language.isoengen_US
dc.publisherS. Karger AGen_US
dc.rightsclosedAccessen_US
dc.subjectAtherosclerotic plaqueen_US
dc.subjectPlateleten_US
dc.subjectAtherothrombosisen_US
dc.subjectStrokeen_US
dc.titlePlatelets, Arterial Thrombosis and Cerebral Ischemiaen_US
dc.typeArtículoen_US
dc.identifier.doi10.1159/000107377-
dc.description.peerreviewedPeer revieweden_US
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