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dc.contributor.authorAranda-Olmedo, Isabel-
dc.contributor.authorMarín, Patricia-
dc.contributor.authorRamos, Juan L.-
dc.contributor.authorMarqués, Silvia-
dc.date.accessioned2008-04-10T12:31:12Z-
dc.date.available2008-04-10T12:31:12Z-
dc.date.issued2006-11-
dc.identifier.citationApplied on Environmental Microbiology 72(11): 7418–7421 (2006)en_US
dc.identifier.issn1098-5336-
dc.identifier.urihttp://hdl.handle.net/10261/3524-
dc.description.abstractThe Pseudomonas putida KT2440 TOL upper pathway is repressed under nonlimiting conditions in cells growing in chemostat with succinate as a carbon source. We show that the ptsN gene product IIANtr participates in this repression. Crc, involved in yeast extract-dependent repression in batch cultures, did not influence expression when cells were growing in a chemostat with succinate at maximum rate.en_US
dc.description.sponsorshipThis work was supported by European Communities grant QLK3- CT-2002-01923, grant BCM 2001-0515 from the Spanish Ministry of Science and Education, and grant VEM2004-08560 from Spanish Ministry of the Environment.en_US
dc.format.extent169893 bytes-
dc.format.mimetypeapplication/pdf-
dc.language.isoengen_US
dc.publisherAmerican Society for Microbiologyen_US
dc.rightsclosedAccessen_US
dc.titleRole of the ptsN Gene Product in Catabolite Repression of the Pseudomonas putida TOL Toluene Degradation Pathway in Chemostat Culturesen_US
dc.typeartículoen_US
dc.identifier.doi10.1128/AEM.01067-06-
dc.description.peerreviewedPeer revieweden_US
dc.relation.publisherversionhttp://dx.doi.org/10.1128/AEM.01067-06-
dc.identifier.pmid16997980-
dc.type.coarhttp://purl.org/coar/resource_type/c_6501es_ES
item.openairetypeartículo-
item.grantfulltextnone-
item.cerifentitytypePublications-
item.openairecristypehttp://purl.org/coar/resource_type/c_18cf-
item.fulltextNo Fulltext-
item.languageiso639-1en-
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