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Por favor, use este identificador para citar o enlazar a este item: http://hdl.handle.net/10261/147132
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dc.contributor.authorAyllón, Nieves-
dc.contributor.authorKocan, Katherine M.-
dc.contributor.authorFuente, José de la-
dc.date.accessioned2017-03-22T11:35:33Z-
dc.date.available2017-03-22T11:35:33Z-
dc.date.issued2015-
dc.identifierdoi: 10.1016/B978-0-12-397169-2.00110-4-
dc.identifierisbn: 978-0-12-397169-2-
dc.identifier.citationMolecular Medical Microbiology 3: 2033-2042 (2015)-
dc.identifier.urihttp://hdl.handle.net/10261/147132-
dc.descriptionChapter 110, Second Edition.-- et al.-
dc.description.abstractAnaplasma phagocytophilum is a rickettsial pathogen transmitted by ixodid ticks. A. phagocytophilum colonises myeloid and non-myeloid cells and causes human granulocytic anaplasmosis – an important disease in the United States, Europe and Asia. Research has illustrated how A. phagocytophilum successfully invades and proliferates inside host cells, causing a systemic disease. Here, we address A. phagocytophilum biology and the underlying mechanisms involved in bacterial pathogenesis and disease development. Clinical features, diagnostics and treatment are also discussed, as well as the contribution of the tick vector for microbial transmission.-
dc.publisherElsevier-
dc.rightsclosedAccess-
dc.subjectVector-borne disease-
dc.subjectTicks-
dc.subjectRickettsial agents-
dc.subjectObligate intracellular bacterium-
dc.titleAnaplasma-
dc.typecapítulo de libro-
dc.identifier.doi10.1016/B978-0-12-397169-2.00110-4-
dc.date.updated2017-03-22T11:35:34Z-
dc.description.versionPeer Reviewed-
dc.language.rfc3066eng-
dc.relation.csicSí-
dc.type.coarhttp://purl.org/coar/resource_type/c_3248es_ES
item.fulltextNo Fulltext-
item.openairecristypehttp://purl.org/coar/resource_type/c_18cf-
item.openairetypecapítulo de libro-
item.cerifentitytypePublications-
item.grantfulltextnone-
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