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Alternative neural circuitry that might be impaired in the development of Alzheimer disease

AuthorsÁvila, Jesús ; Perry, George; Strange, Bryan A.; Hernández Pérez, Félix
Issue Date23-Apr-2015
PublisherFrontiers Media
CitationFrontiers in Neuromorphic Engineering 9: 145 (2015)
AbstractIt is well established that some individuals with normal cognitive capacity have abundant senile plaques in their brains. It has been proposed that those individuals are resilient or have compensation factors to prevent cognitive decline. In this comment, we explore an alternative mechanism through which cognitive capacity is maintained. This mechanism could involve the impairment of alternative neural circuitry. Also, the proportion of molecules such as Aβ or tau protein present in different areas of the brain could be important.
Identifiersdoi: 10.3389/fnins.2015.00145
issn: 1662-453X
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