2024-03-29T10:12:25Zhttp://digital.csic.es/dspace-oai/requestoai:digital.csic.es:10261/819712018-09-12T09:19:03Zcom_10261_15com_10261_6col_10261_268
A passive repression mechanism that hinders synergic transcriptional activation by heat shock factors involved in sunflower seed longevity
Tejedor-Cano, Javier
Carranco, Raúl
Personat, José María
Prieto-Dapena, P.
Almoguera, Concepción
Espinosa-Vázquez, José Manuel
Jordano, Juan
Different genetic programs that are active during late stages of zygotic
embryogenesis are involved in seed phenotypes as longevity, basal
thermotolerance, and desiccation tolerance. In sunflower, Heat Shock Factor A9
(HaHSFA9) controls one of such programs, the HaHSFA9 program. Overexpression
of HaHSFA9 in seeds of transgenic tobacco showed the
involvement of this transcription factor in basal thermotolerance and longevity;
HaHSFA9 activated a genetic program that includes specific small Heat Shock
Protein (sHSP) genes, which are expressed mainly (or exclusively) during
zygotic embryogenesis in seeds (Prieto-Dapena et al., 2006). We also used an
active-repressor dominant-negative (DN) form of HaHSFA9, and thus we
obtained a substantial reduction of both the accumulation of specific sHSPs and
of seed longevity (Tejedor-Cano et al., 2010).
2013-09-13T07:41:12Z
2013-09-13T07:41:12Z
2014-01
artículo
Molecular Plant: 7(1) 256–259 (2014)
1674-2052
http://hdl.handle.net/10261/81971
http://dx.doi.org/doi:10.1093/mp/sst117
10.1093/mp/sst117
1752-9867
eng
http://dx.doi.org/doi:10.1093/mp/sst117
openAccess
Oxford University Press