2024-03-28T09:51:58Zhttp://digital.csic.es/dspace-oai/requestoai:digital.csic.es:10261/1241002021-12-28T16:34:05Zcom_10261_79com_10261_1col_10261_332
00925njm 22002777a 4500
dc
Ávila, Jesús
author
Perry, George
author
Strange, Bryan A.
author
Hernández Pérez, Félix
author
2015-04-23
It is well established that some individuals with normal cognitive capacity have abundant senile plaques in their brains. It has been proposed that those individuals are resilient or have compensation factors to prevent cognitive decline. In this comment, we explore an alternative mechanism through which cognitive capacity is maintained. This mechanism could involve the impairment of alternative neural circuitry. Also, the proportion of molecules such as Aβ or tau protein present in different areas of the brain could be important.
Frontiers in Neuromorphic Engineering 9: 145 (2015)
http://hdl.handle.net/10261/124100
10.3389/fnins.2015.00145
http://dx.doi.org/10.13039/501100003751
http://dx.doi.org/10.13039/100012818
25954151
Alternative neural circuitry that might be impaired in the development of Alzheimer disease